Syllabus Edition
First teaching 2025
First exams 2027
Cortisol , Stress & Obesity (DP IB Psychology): Revision Note
The cortisol-awakening response (CAR)
Cortisol is a hormone produced in the adrenal cortex via activation of the hypothalamus-pituitary-adrenal (HPA) axis when the body is exposed to stress
The immune system (mainly white blood cells) defends the body against harmful antigens such as bacteria, viruses, and cancerous cells
Cortisol normally helps by regulating the immune system and suppressing inflammation, which is usually beneficial
However, under chronic stress, excess cortisol builds up in the bloodstream:
This leads to over-suppression of the immune system
As a result, the body’s resistance to illness is lowered
Stress does not directly cause infection, but it reduces the immune system’s ability to fight antigens, making infections more likely
This explains why many people often develop colds or minor illnesses during exam periods, when stress levels (and cortisol) are high
Obesity & stress
Stress can lead to obesity, and obesity can increase stress levels – it is a two-way relationship
Stress affects self-regulation
Stress reduces people’s ability to control their behaviour (e.g., resisting fatty, sugary foods)
Stress disrupts eating-related hormones and peptides:
Leptin – signals fullness to the brain
Ghrelin – signals hunger
Neuropeptide Y – regulates appetite
Disruption to these hormones makes overeating more likely under stress
Research support for cortisol, stress & obesity
Kumar et al. (2022)
Aim:
To examine the link between stress and obesity
Method:
A literature review of journal articles on stress, cortisol and obesity published between 2000 and 2020
Results:
Obesity treatment is more effective if stress is targeted
Stress is a major factor in obesity (though biological and sociocultural factors also play a role)
Research shows people eat more under stress due to the CAR
Many people self-medicate with comfort food during stress, which increases BMI over time
Recognising and addressing stress-eating is key to breaking the CAR–stress–obesity cycle
Conclusion:
Stress and obesity are closely connected, which means that they should be studied and treated together
Evaluation of cortisol, stress & obesity
Strengths
Strong biological evidence for the link between cortisol and stress makes the obesity-related findings convincing
Stress management strategies (e.g., walking, meditation, taking a warm bath) are accessible and practical for most people who require to a solution to their obesity
Limitations
Tackling stress-related eating is difficult because eating is pleasurable, making behaviour change hard to sustain
Individual differences are not accounted for, as not everyone eats more when stressed; some lose their appetite instead
This limits the explanation, as stress does not affect all people in the same way
Link to concepts
Measurement
Conducting a literature review is a good way of gaining a comprehensive appreciation of a complex issue such as stress-related obesity
Drawing from a range of cognitive, sociocultural and biological research, it is possible to gain a holistic understanding of the issue rather than focusing on one narrow and possibly limited explanation of a health problem
Bias
Confirmation bias is a potential threat to validity with a literature review
The researcher must practise reflexivity to ensure that their own biases, prejudices and preconceived ideas do not lead to them only selecting data which supports their research hypothesis/objective
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